Medical School

Rheumatic and Autoimmune Diseases

Department of Medicine

Erik J. Peterson, MD

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Academic Title

Associate Professor of Medicine

Clinical Title

Rheumatologist

Specializations

  • Arthritis and Autoimmune Disorders (Rheumatology)

Practices

  • Rheumatology Clinic

Education

Medical School
University of Minnesota
Residency
University of Colorado, Denver (Internal Medicine)
Fellowship
University of Iowa Hospital and Clinics (Rheumatology)

Administrative Contact

General Information
E: Rheum
P: (612) 624-5346
F: (612) 624-0600

Clinic Appointments
P: (612) 626-6100
W: University of Minnesota Physicians

Mailing Address

Division of Rheumatic and Autoimmune Diseases
420 Delaware Street SE, MMC 108
Minneapolis, MN 55455
 

Bio Statement

Dr. Erik Peterson joined the division of Rheumatic and Autoimmune Diseases in July, 2002. He is a member of the interdisciplinary Center for Immunology and its Autoimmunity Program, as well as a member of the Cancer Center. He is an immunologist with a scientific interest in those molecules that regulate the development and function of the immune system and prevent or promote the development of autoimmunity. He is also practices general rheumatology and participates in Resident and Fellow teaching.

Research in the Peterson laboratory
Dr. Erik Peterson has strong interests in the molecular underpinnings of autoimmune diseases, including rheumatoid arthritis, lupus, and myositis. His laboratory utilizes genetic, biochemical, and primary human sample-based approaches to investigating the mechanisms whereby recently identified “risk” genes predispose to development of autoimmune disease. Dr. Peterson's group recently identified a role for Ptpn22, a potent “risk” gene for many autoimmune diseases, in the promotion of toll-like receptor signaling and type 1 interferon production. He is currently investigating the role of Ptpn22 in myeloid cell functions in systemic lupus and in responses to immunization, and is characterizing the molecular mechanism of Ptpn22 promotion of type 1 interferon signals.

 

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  • Last modified on December 26, 2013